Research progress of mitophagy in chronic cerebral ischemia

Published on August 3, 2023

Imagine your brain is a bustling city, with blood vessels serving as its highways. But what happens when those highways start to narrow and traffic slows down? That’s exactly what chronic brain ischemia (CCI) does – it restricts the flow of blood to your brain, leading to symptoms like headaches, dizziness, and even stroke. But fear not! Recent research has uncovered an exciting new mechanism called mitophagy that could hold the key to mitigating the effects of CCI. Mitophagy is like a cleaning crew for your brain cells, disposing of damaged or dysfunctional mitochondria to maintain a healthy energy supply. By understanding how mitophagy is induced in CCI – through pathways like ATP synthesis disorder, oxidative stress injury, and Ca2+ homeostasis disorder – scientists can devise targeted therapies to regulate this process and potentially alleviate the impact of CCI. So, hop on over to the article linked below to delve deeper into this groundbreaking research and discover how mitophagy could unlock new treatments for chronic brain ischemia!

Chronic cerebral ischemia (CCI), a condition that can result in headaches, dizziness, cognitive decline, and stroke, is caused by a sustained decrease in cerebral blood flow. Statistics show that 70% of patients with CCI are aged > 80 years and approximately 30% are 45–50 years. The incidence of CCI tends to be lower, and treatment for CCI is urgent. Studies have confirmed that CCI can activate the corresponding mechanisms that lead to mitochondrial dysfunction, which, in turn, can induce mitophagy to maintain mitochondrial homeostasis. Simultaneously, mitochondrial dysfunction can aggravate the insufficient energy supply to cells and various diseases caused by CCI. Regulation of mitophagy has become a promising therapeutic target for the treatment of CCI. This article reviews the latest progress in the important role of mitophagy in CCI and discusses the induction pathways of mitophagy in CCI, including ATP synthesis disorder, oxidative stress injury, induction of reactive oxygen species, and Ca2+ homeostasis disorder, as well as the role of drugs in CCI by regulating mitophagy.

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