Oxidative stress in Wernicke’s encephalopathy

Published on May 16, 2023

Wernicke’s encephalopathy is like a delicate machine that malfunctions when it lacks an essential component, vitamin B1. This severe disease causes chaos in the brain, resulting in mental disorders and physical impairments. Recent research suggests that oxidative stress, like the wear and tear on a machine, plays a pivotal role in damaging organs and exacerbating the condition. In this review, we explore the far-reaching effects of oxidative stress on various organ systems in individuals with Wernicke’s encephalopathy, such as the liver, intestines, and of course, the brain. By strengthening the body’s antioxidant defenses and addressing vitamin B1 deficiency, we may have found a promising avenue for treating this life-threatening ailment.

Wernicke’s encephalopathy (WE) is a severe life-threatening disease that occurs due to vitamin B1 (thiamine) deficiency (TD). It is characterized by acute mental disorder, ataxia, and ophthalmoplegia. TD occurs because of the following reasons: insufficient intake, increased demand, and long-term drinking due to corresponding organ damage or failure. Recent studies showed that oxidative stress (OS) can damage organs and cause TD in the brain, which further leads to neurodegenerative diseases, such as WE. In this review, we discuss the effects of TD caused by OS on multiple organ systems, including the liver, intestines, and brain in WE. We believe that strengthening the human antioxidant system and reducing TD can effectively treat WE.

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