Navigating the Neuronal Battlefield: Controlling Microglial Polarization in TBI

Published on August 1, 2022

In the treacherous world of traumatic brain injury (TBI), microglia are like warriors defending the central nervous system (CNS). These specialized troops, known as macrophages, are divided into two factions: the inflammation-promoting M1 and the inflammation-inhibiting M2. Steering microglia toward the desired polarization is crucial for TBI treatment. Elucidating the intricate signal pathways involved, such as TLR-4/NF-κB, JAK/STAT, HMGB1, MAPK, and PPAR-γ, helps us identify potential pharmaceutical allies. Fingolimod, minocycline, Tak-242, erythropoietin (EPO), and CSF-1 emerge as candidates that target these pathways. Exploring their impact on controlling microglial polarization could revolutionize TBI therapy. Dive into the research to uncover the secrets of this cerebral battlefield!

Traumatic brain injury (TBI) is a serious disease that threatens life and health of people. It poses a great economic burden on the healthcare system. Thus, seeking effective therapy to cure a patient with TBI is a matter of great urgency. Microglia are macrophages in the central nervous system (CNS) and play an important role in neuroinflammation. When TBI occurs, the human body environment changes dramatically and microglia polarize to one of two different phenotypes: M1 and M2. M1 microglia play a role in promoting the development of inflammation, while M2 microglia play a role in inhibiting inflammation. How to regulate the polarization direction of microglia is of great significance for the treatment of patients with TBI. The polarization of microglia involves many cellular signal transduction pathways, such as the TLR-4/NF-κB, JAK/STAT, HMGB1, MAPK, and PPAR-γ pathways. These provide a theoretical basis for us to seek therapeutic drugs for the patient with TBI. There are several drugs that target these pathways, including fingolimod, minocycline, Tak-242 and erythropoietin (EPO), and CSF-1. In this study, we will review signaling pathways involved in microglial polarization and medications that influence this process.

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